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21. Adan RA, Van Leeuwen FW, Sonnemans MA, Brouns M, Hoffman G, Verbalis JG, Burbach JP. Rat oxytocin receptor in brain, pituitary, mammary gland, and uterus: partial sequence and immunocytochemical localization. Endocrinology 1995; 136: 40224028. Wu WX, Verbalis JG, Hoffman GE, Derks JB, Nathanielsz PW. Characterization of oxytocin receptor expression and distribution in the pregnant sheep uterus. Endocrinology 1996; 137: 722728. Schrey MP, Cornford PA, Read AM, Steer PJ. A role for phosphoinositide hydrolysis in human uterine smooth muscle during parturition. J Obstet Gynecol 1988; 159: 964970. Ueno N, Zhao Y, Zhang L, Longo LD. High altitude-induced changes in 1-adrenergic receptors and Ins 1, 4, 5 ; P3 responses in cerebral arteries. J Physiol 1997; 272: R669R674. 25. Hu XQ, Longo LD, Gilbert RD, Zhang L. Effects of long-term high FIG. 2. Time course of effects of oxytocin and insulin on accumulation of IPS in fat cells. Fat cells were incubated with , [SH]inositol 10 p ~ pCi ml ; for 2 h. During the last 30 min, LiCl 5 mM ; was added. At the end of a 2-h incubation, the medium was removed, and the cells werewashed three times. They were then incubated for 30 min in the presence of LiCl 10 mM ; before the addition of oxytocin 3.3 p M ; or insulin 10 nM ; for the indicated times. Accumulation of IPS was measured as detailed under "Experimental Procedures." Results are expressed as the means + S.E. of three to five determinations obtained from two different experiments. Apparently, a decline in circulating estro­ gen and prolactin, an increase in oxytocin and possibly prolactin ; , cervical stimulation and the presence of a small puppy with a fore­ shortened face and wet with amniotic fluid are all factors involved in establishing the bitch's maternal behavior. 443 RELEASE OF VASOPRESSIN AND OXYTOCIN present or because its binding capacity is different from that in glands of adult rats. The actual diffusion of the hormones across the cell membrane would then be achieved or be accompanied by a shift of ions, similar to that observed after addition of ouabain to the incubation medium of pituitary glands from either adult or new-born rats. Thus, whereas in the hypophysis of adult animals, the release of hormones involves their liberation from a protein carrier before their secretion, in glands of newborn animals the release of hormones may be explained simply by their diffusion across the cell membrane. This would account for the observations that the secretion of posterior pituitary glands of neonates does not appear to be influenced by exogenous stimuli in the same way as neurohypophysial glands of adults.
Harry Bass MD Las Cruces ; , Delegate; Steven Kanig MD Albuquerque ; , Delegate; Allan Haynes, Jr. MD Clovis ; , Alternate Delegate; Warren Heffron MD Albuquerque ; , Alternate Delegate; Stephen Lucero MD Santa Fe ; , President NMMS; James Tryon MD Albuquerque ; , President-Elect NMMS; Marion Jordan MD Albuquerque ; , Delegate, American College of Nuclear Physicians; Timothy Lopez MD Santa Fe ; , Delegate, American Society of Hematology; Barbara McAneny MD Albuquerque ; , Delegate, Amercian Society of Clinical Oncology; John Seibel MD Albuquerque ; , Delegate, American Association of Clinical Endocrinologists; Steven Tolber MD Albuquerque ; , Alternate Delegate, American Academy of Asthma, Allergy and Immunology; Eric Anderson MD Santa Fe ; , Young Physicians Section.

Key to the Group's success is maximising the number of instrument placements in order to generate high margin consumable revenues for future years. The graph clearly demonstrates the recent successes in instrument sales. Moser, K. M., Perry, R. B., and Luchsinger, P. C.: Cardiopulmonary Consequences of Pyrogen-induced Hyperpyrexia in Man. J. Clin. Invest. 42: 626 May ; , 1963. Intravenous injections of a lipopolysaccharide extract of gram-negative bacilli were given to 10 men free from obvious cardiopulmonary disease in order to provoke a pyrogenic response. Four clinical phases followed the injections: prodrome period of stable temperature chill rising temperature flush peak temperature defervescence falling temperature ; . No change in monitored cardiovascular functions occurred during phase 1. During phases 2, 3, and 4 there were increases in oxygen consumption, minute and alveolar ventilation, respiratory rate, and heart rate. During phase 3, the cardiac output increased, "total pulmonary resistance" was unchanged, and systemic vascular resistance decreased. These changes in cardiovascular function induced by a pyrogen could be enough to cause cardiac failure in patients with impaired reserve and parnate.
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